Author

M. Philips

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Cancer Biology· 1

Signature Assays, Organisms & Methods

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Organism:human· 1 Approach:statistical model· 1 Research Focus:variant calling· 1 Research Focus:variant effect prediction· 1 Assay:whole-exome sequencing· 1

Frequent Co-Authors

A. Greenstein· 1 shared C. Stender· 1 shared J. P. Townsend· 1 shared S. G. Gaffney· 1 shared V. L. Cannataro· 1 shared Z.-M. Zhao· 1 shared

Activity by Year

2017-2017 · peak 1/yr

Co-Author Collaboration Network

6 top collaborators · 15 mutual co-author links

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1 shared paperCancer Biology

A. Greenstein

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1 paper

Recent·Most cited

The likelihood of heterogeneity or additional mutation in KRAS or associated oncogenes to compromise targeting of oncogenic KRAS G12C

V. L. Cannataro, S. G. Gaffney, C. Stender, Z.-M. Zhao, M. Philips, A. Greenstein, J. P. Townsend

The study finds no evidence of resistance-conferring mutations in KRAS or downstream genes in KRAS G12C positive tumors at treatment time, and predicts BRAF mutations as likely resistance mechanisms with existing therapies.

bioRxiv · Jun 13, 2017Cancer Biology