Author

Takuhiro Kobayashi

The University of Osaka

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Organism:mouse· 1 Approach:statistical model· 1

Frequent Co-Authors

Emiko Tanaka IsomuraiD· 1 shared Kazuaki MiyagawaiD· 1 shared Masayuki OnoiD· 1 shared Soju SekiiD· 1 shared Sou Kawata· 1 shared Susumu TanakaiD· 1 shared Takahide ItokazuiD· 1 shared Toru YamamotoiD· 1 shared

Activity by Year

2026-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperNeuroscience

Emiko Tanaka IsomuraORCID

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1 paper

Recent·Most cited

Sensory neuron dysfunction and hyperexcitability in dorsal root ganglia at disease onset in the SOD1G93A mouse model of ALS.

輝西浦, Soju Seki, Takuhiro Kobayashi, Yoshihiro Kitaoka, Sou Kawata, Toru Yamamoto, Kazuaki Miyagawa, Toshihiro Uchihashi, Yosuke Ohtake, Takahide Itokazu, Toshihide Yamashita, Masayuki Ono, Yusuke Yokota, Emiko Tanaka Isomura, Susumu Tanaka

The paper identifies early molecular, structural, and functional changes in sensory neurons in the SOD1G93A mouse model of ALS, suggesting sensory neuron hyperexcitability as a potential therapeutic target.

bioRxiv · Sep 1, 2026 · PreprintNeuroscience