Author

Azmal Syed Ali

German Cancer Research CenterPrimary ORCID profile (0000-0003-3024-9379 · 1 paper)Unclaimed ORCID profile · follow to get notified of new preprints

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Azmal Syed AliiD1 paper · Division of Proteomics of Stem Cell and Cancer, German Cancer Research Center (DKFZ), Heidelberg, Germany;

Biology Disciplines

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Neuroscience· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

Baran E. GüleriD· 1 shared Celine E. de Esch· 1 shared Christian P. Schaaf· 1 shared Derek J.C. TaiiD· 1 shared Freya Herrmann-Sim· 1 shared Jannis BueckingiD· 1 shared Jeroen KrijgsveldiD· 1 shared Katrin HinderhoferiD· 1 shared

Activity by Year

2026-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperNeuroscience

Baran E. GülerORCID

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1 paper

Recent·Most cited

Multi-omics profiling reveals MAGEL2-driven defects in human corticogenesis shared across Prader-Willi and Schaaf-Yang syndromes.

Jannis Buecking, Baran E. Güler, Michael Eibl, Azmal Syed Ali, Tobias Walczuch, Tobias Beschauner, Susanne Theiß, Melanie Spanjaard, Katrin Hinderhofer, Freya Herrmann-Sim, Celine E. de Esch, Derek J.C. Tai, Michael E. Talkowski, Jeroen Krijgsveld, Christian P. Schaaf, Magdalena Laugsch

This study identifies MAGEL2-driven defects in human corticogenesis shared between Prader-Willi and Schaaf-Yang syndromes using multi-omics profiling of induced neurons.

bioRxiv · May 5, 2026 · PreprintNeuroscience