B. Ashby

Coevolution of age-structured tolerance and virulence

This study models how age-specific tolerance and virulence coevolve and finds that age structure influences coevolutionary dynamics and virulence selection.

Separation of Evolutionary Timescales in Coevolving Species

The paper introduces a method to determine convergence stability in coevolutionary processes where one species evolves much faster than the other, reducing computational complexity by separating timescales.

Hyperparasitism and the evolution of parasite virulence

This study uses a host-parasite-hyperparasite model to show hyperparasites can drive changes in parasite virulence and induce virulence diversification, with potential implications for biocontrol strategies.

Tolerance-conferring defensive symbionts and the evolution of parasite virulence

This paper shows that introducing tolerance-conferring defensive symbionts can lead to increased parasite virulence and long-term host population decline, suggesting they are poor for biocontrol.

The Evolution of the Age of Onset of Resistance to Infectious Disease

The paper investigates how the timing of costs associated with developing disease resistance affects its evolutionary onset and finds that timing significantly influences whether resistance develops early or late.

The evolution of age-specific resistance to infectious disease

This study uses a mathematical model to show how trade-offs between life-history traits affect the evolution of age-specific resistance to infectious disease, finding that juvenile resistance is often lower due to trade-offs with adult reproduction.

Social information use shapes the coevolution of sociality and virulence

The paper models how sociality and parasite virulence coevolve based on the interplay between social information transmission and infection risk.

When does parasitism maintain sex in the absence of Red Queen Dynamics?

This study uses an eco-evolutionary model to show that population density and parasite virulence affect the coexistence of sex and asex in the absence of Red Queen Dynamics.

How do biases in sex ratio and disease characteristics affect the spread of sexually transmitted infections?

This study uses mathematical modeling to show that skewed sex ratios and sex-biased disease characteristics influence STI prevalence differently in males and females.

An inordinate fondness for species with intermediate dispersal abilities

The paper shows that species with intermediate dispersal abilities are more likely to achieve high species richness due to eco-evolutionary effects of dispersal on speciation.

Antagonistic coevolution between hosts and sexually transmitted infections

This study models host-STI coevolution, showing mate choice evolves despite ephemeral mating and acute infections, constraining virulence and leading to fluctuating selection under specific conditions.