Author

Brad A. Godfrey

Michigan Medicine

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Systems Biology· 2

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:differential expression· 2 Organism:human· 2 Approach:statistical model· 2 Research Focus:benchmarking· 1 Research Focus:cell type annotation· 1

Frequent Co-Authors

Felix H. EichingeriD· 2 shared Matthias KretzleriD· 2 shared Richard A. LafayetteiD· 2 shared Sean F. EddyiD· 2 shared Viji NairiD· 2 shared Alessia FornoniiD· 1 shared Alicia M. NeuiD· 1 shared Ambarish M. Athavale· 1 shared

Activity by Year

2018-2018 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 41 mutual co-author links

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2 shared papersSystems Biology

Felix H. EichingerORCID

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2 papers

Recent·Most cited

Redefining Nephrotic Syndrome in Molecular Terms: Outcome-associated molecular clusters and patient stratification with noninvasive surrogate biomarkers

Laura Mariani, Sean F. Eddy, Sebastian Martini, Felix H. Eichinger, Brad A. Godfrey, Viji Nair, Sharon G. Adler, Gerry B. Appel, Ambarish M. Athavale, Laura M. C. Barisoni, Elizabeth Brown, Dan C. Cattran, Katherine M. Dell, Vimal Kumar Derebail, Fernando Custodio Fervenza, , Matthias Kretzler

The paper identifies high-risk nephrotic syndrome patients through transcriptome analysis and develops non-invasive biomarkers to predict disease progression.

bioRxiv · Sep 27, 2018 · PreprintSystems Biology

Inflammatory and JAK-STAT Pathways as Shared Molecular Targets for ANCA-Associated Vasculitis and Nephrotic Syndrome

Sean F. Eddy, Viji Nair, Laura H. Mariani, Felix H. Eichinger, John Hartman, Huateng Huang, Hemang M. Parikh, Jaclyn Taroni, Maja T. Lindenmeyer, Wenjun Ju, Casey S. Greene, Peter C. Grayson, Brad A. Godfrey, Clemens David Cohen, Matthew Gordon Sampson, , Matthias Kretzler

The paper identifies shared molecular pathways between ANCA-associated vasculitis and nephrotic syndrome, highlighting JAK-STAT signaling as a potential therapeutic target.

bioRxiv · Sep 27, 2018 · PreprintSystems Biology