Author

John Hartman

University of Michigan

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Systems Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Assay:organoid / primary culture· 1

Frequent Co-Authors

Felix H. EichingeriD· 2 shared Matthias KretzleriD· 2 shared Viji NairiD· 2 shared Akihiro MinakawaiD· 1 shared Anja M. BillingiD· 1 shared Brad A. Godfrey· 1 shared Bradley GodfreyiD· 1 shared Casey S. GreeneiD· 1 shared

Activity by Year

2018-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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2 shared papersCell Biology

Felix H. EichingerORCID

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Author profile →

2 papers

Recent·Most cited

Dapagliflozin mitigates hypoxia-induced metabolic stress, kidney tubular cell death and fibrosis

Akihiro Minakawa, Jamal El Saghir, Céline C. Berthier, Moritz Lassé, Soumya Maity, Judy J. Baek, Damian Fermin, Anja M. Billing, Chenchen He, Felix H. Eichinger, John Hartman, Viji Nair, Matthew Fischer, Virginia Vega-Warner, Fadhl Alakwaa, , Jennifer L. Harder

This study shows dapagliflozin reduces hypoxia-induced metabolic stress, cell death, and fibrosis in kidney organoids derived from human pluripotent stem cells.

bioRxiv · Jan 14, 2026 · PreprintCell Biology

Inflammatory and JAK-STAT Pathways as Shared Molecular Targets for ANCA-Associated Vasculitis and Nephrotic Syndrome

Sean F. Eddy, Viji Nair, Laura H. Mariani, Felix H. Eichinger, John Hartman, Huateng Huang, Hemang M. Parikh, Jaclyn Taroni, Maja T. Lindenmeyer, Wenjun Ju, Casey S. Greene, Peter C. Grayson, Brad A. Godfrey, Clemens David Cohen, Matthew Gordon Sampson, , Matthias Kretzler

The paper identifies shared molecular pathways between ANCA-associated vasculitis and nephrotic syndrome, highlighting JAK-STAT signaling as a potential therapeutic target.

bioRxiv · Sep 27, 2018 · PreprintSystems Biology