Author

D. J. Mullen

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Organism:human· 2 Approach:statistical model· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

C. N. Marconett· 2 shared A. Castaldi· 1 shared A. L. Epstein· 1 shared A. L. Ryan· 1 shared B. Zhou· 1 shared H. Shen· 1 shared J. Carpten· 1 shared J. Castillo· 1 shared

Activity by Year

2022-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersCancer Biology

C. N. Marconett

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2 papers

Recent·Most cited

LINC00261 expression reduces mutation accumulation while conferring resistance to cisplatin and altering the tumor-immune microenvironment in lung adenocarcinoma

J. Castillo, T. Xue, S. Joseph, M. Norwood, D. J. Mullen, S. Berrellez, S. James, A. L. Epstein, T. McEachron, C. N. Marconett

This study identifies LINC00261 as a gene that reduces mutation accumulation, confers cisplatin resistance, and alters the tumor-immune microenvironment in lung adenocarcinoma.

bioRxiv · Mar 2, 2023 · PreprintCancer Biology

Alveolar epithelial type 1 cells serve as a cell of origin for lung adenocarcinoma with distinct molecular and phenotypic presentation

M. Yang, H. Shen, P. Flodby, M. Koss, R. Bassiouni, Y. Liu, T. Jashashvilli, T. R. Stueve, D. J. Mullen, A. L. Ryan, J. Carpten, A. Castaldi, W. D. Wallace, B. Zhou, MD, Z. Borok, C. N. Marconett

This study identifies alveolar epithelial type 1 cells as a potential cell of origin for lung adenocarcinoma, showing distinct molecular and histological features based on cell of origin.

bioRxiv · Oct 29, 2022 · PreprintCancer Biology