Author

E. Cabiscol

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1

Frequent Co-Authors

A. Sanz-Alcazar· 2 shared F. Delaspre· 2 shared J. Ros· 2 shared J. Tamarit· 2 shared M. Medina-Carbonero· 2 shared E. Britti· 1 shared G. Hernandez· 1 shared L. Oliveira· 1 shared

Activity by Year

2021-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 41 mutual co-author links

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2 shared papersBiochemistry

A. Sanz-Alcazar

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2 papers

Recent·Most cited

SYSTEMIC FRATAXIN DEFICIENCY CAUSES TISSUE-DEPENDENT IRON HOMEOSTASIS ALTERATIONS: IMPLICATIONS FOR FRIEDREICH ATAXIA

M. Pazos-Gil, M. Medina-Carbonero, A. Sanz-Alcazar, M. Portillo-Carrasquer, L. Oliveira, G. Hernandez, M. Sanchez, F. Delaspre, E. Cabiscol, J. Ros, J. Tamarit

This study shows that frataxin deficiency causes tissue-specific iron homeostasis changes in FXNI151F mice, with brain iron overload and cardiac iron deficiency responses.

bioRxiv · Jul 5, 2024 · PreprintBiochemistry

Biochemical alterations precede neurobehavioral deficits in a novel mouse model of Friedreich ataxia

M. Medina-Carbonero, A. Sanz-Alcazar, E. Britti, F. Delaspre, E. Cabiscol, J. Ros, J. Tamarit

This study characterizes a new mouse model of Friedreich ataxia with low frataxin levels and early biochemical changes preceding neurological symptoms.

bioRxiv · Apr 5, 2021 · PreprintBiochemistry