Author

E. Ulbricht

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biophysics· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Approach:biophysical modeling· 1 Research Focus:development & morphogenesis· 1 Assay:imaging· 1

Frequent Co-Authors

A. V. Taubenberger· 2 shared A. Jagielska· 1 shared A. Lahola-Chomiak· 1 shared D. Alves-Afonso· 1 shared D. Eberle· 1 shared G. Fodelianaki· 1 shared J. Guck· 1 shared J. Lattner· 1 shared

Activity by Year

2018-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersBiophysics

A. V. Taubenberger

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2 papers

Recent·Most cited

Self-propagating wave drives noncanonical antidurotaxis of skull bones in vivo

Y. Dang, J. Lattner, A. Lahola-Chomiak, D. Alves-Afonso, E. Ulbricht, A. V. Taubenberger, S. Rulands, J. Tabler

The paper identifies a self-propagating motion mechanism in skull bone morphogenesis driven by mechanical feedback between stiffness and cell fate, termed noncanonical antidurotaxis.

bioRxiv · Jul 10, 2023 · PreprintBiophysics

Acute but not inherited demyelination in mouse models leads to brain tissue stiffness changes

D. Eberle, G. Fodelianaki, T. Kurth, A. Jagielska, S. Moellmert, E. Ulbricht, K. Wagner, A. V. Taubenberger, N. Traeber, J.-C. Escolano, R. Franklin, K. J. Van Vliet, J. Guck

This study shows that acute demyelination reduces brain tissue stiffness, while inherited demyelination does not, potentially affecting remyelination in mouse models.

bioRxiv · Nov 5, 2018 · PreprintBiophysics