PTBP1 promotes cardiac hypertrophy anddiastolic dysfunction by modulating alternativesplicing
Carlos Martí‐Gómez, Javier Larrasa-Alonso, Marina M. López-Olañeta, María Villalba‐Orero, Pablo García‐Pavía, Fátima Sánchez‐Cabo, Enrique Lara‐Pezzi
The paper investigates alternative splicing (AS) in cardiac pathophysiology, identifying PTBP1 as a key regulator of AS changes that drive cardiac hypertrophy and diastolic dysfunction.
bioRxiv · Jun 30, 2020 · PreprintBioinformatics