Author

Paul J.D. Winyard

University College London

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Physiology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:genetic perturbation / CRISPR· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

Christopher J. RowaniD· 1 shared David Andrew LongiD· 1 shared Georgie Stagg· 1 shared Jennifer C. ChandleriD· 1 shared Karen L. PriceiD· 1 shared Laura WilsoniD· 1 shared Lauren G. RusselliD· 1 shared Maria Kolatsi‐JoannouiD· 1 shared

Activity by Year

2025-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperPhysiology

Christopher J. RowanORCID

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1 paper

Recent·Most cited

Reversal of elevated Gli3 in Autosomal Recessive Polycystic Kidney Disease does not alter cystogenesis

Lauren G. Russell, Maria Kolatsi‐Joannou, Laura Wilson, Jennifer C. Chandler, Nuria Perretta‐Tejedor, Georgie Stagg, Karen L. Price, Christopher J. Rowan, Tessa Crompton, Norman D. Rosenblum, Paul J.D. Winyard, David Andrew Long

This study investigates the role of elevated Gli3 in ARPKD and finds that reducing Gli3 levels does not affect cyst formation in mouse or human models.

Physiological Reports · Jan 1, 2025Peer-reviewedPhysiology