Author

S. Willis

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Evolutionary Biology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:chromatin & epigenetic regulation· 3 Approach:statistical model· 2 Assay:ATAC-seq· 1 Research Focus:cell type annotation· 1 Assay:DNA methylation· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Organism:multiple species· 1

Frequent Co-Authors

J. Masel· 2 shared A. L. Wheeler· 1 shared C. Weibel· 1 shared H. McShea· 1 shared J. E. James· 1 shared K. Cooper· 1 shared S. E. Hanley· 1 shared

Activity by Year

2018-2023 · peak 1/yr

Co-Author Collaboration Network

7 top collaborators · 11 mutual co-author links

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2 shared papersEvolutionary Biology

J. Masel

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3 papers

Recent·Most cited

The protein domains of vertebrate species in which selection is more effective have greater intrinsic structural disorder

C. Weibel, A. L. Wheeler, J. E. James, S. Willis, H. McShea, J. Masel

The paper introduces a new Codon Adaptation Index (CAIS) to measure selection effectiveness in vertebrates and shows that more adapted species have protein domains with higher intrinsic structural disorder.

bioRxiv · Mar 2, 2023Evolutionary Biology

Snx4-mediated nucleophagy targets transcription factors controlling ATG gene expression

K. Cooper, S. E. Hanley, S. Willis

The paper identifies a novel nucleophagy pathway mediated by Snx4 that targets transcription factors regulating ATG gene expression, enabling fine-tuned autophagic responses.

bioRxiv · May 27, 2020Cell Biology

Gene birth contributes to structural disorder encoded by overlapping genes

S. Willis, J. Masel

The paper investigates how gene birth contributes to structural disorder in overlapping genes, finding that recent de novo gene birth explains 32% of the increased intrinsic structural disorder in viral genes.

bioRxiv · Jun 13, 2018Evolutionary Biology