Author

Samuel T Moss

University of Leicester

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Genetic & Genomic Medicine· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:GWAS / association mapping· 1 Organism:human· 1 Approach:statistical model· 1

Frequent Co-Authors

Abril G. IzquierdoiD· 1 shared Alison E. JohniD· 1 shared Amy Stockwell· 1 shared Ann Millar· 1 shared Ayodeji AdegunsoyeiD· 1 shared Beatriz Guillén‐GuíoiD· 1 shared Brian L. YaspaniD· 1 shared Carl ReynoldsiD· 1 shared

Activity by Year

2026-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperGenetic & Genomic Medicine

Abril G. IzquierdoORCID

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1 paper

Recent·Most cited

Contribution of dominant and recessive model effects to the genetic architecture of Idiopathic Pulmonary Fibrosis

Tamara Hernández-Beeftink, Lauren J. Donoghue, Abril G. Izquierdo, Samuel T Moss, Daniel Chin, Beatriz Guillén‐Guío, Konain Fatima Bhatti, Simon Cameron Biddie, Nick R. G. Shrine, Richard J. Packer, Ayodeji Adegunsoye, Helen Booth, CleanUP-IPF Investigators of the Pulmonary Trials Cooperative, William A. Fahy, Tasha E. Fingerlin, , Louise V. Wain

This study performs GWAS of IPF using dominant and recessive models, identifying five new genome-wide significant signals and genes, including PMF1 and EPN3, and observing increased PMF1 expression in IPF patients.

medRxiv · Feb 19, 2026 · PreprintGenetic & Genomic Medicine