Author

Sawsan Mahmoud

Université du Québec à Trois-Rivières

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 3

Signature Assays, Organisms & Methods

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Assay:imaging· 3 Organism:human· 2 Approach:statistical model· 2 Research Focus:benchmarking· 1 Research Focus:cell type annotation· 1 Research Focus:clustering· 1 Approach:deep learning· 1 Research Focus:differential expression· 1

Frequent Co-Authors

Josefina MaranzanoiD· 3 shared Mahsa DadariD· 3 shared Maryna ZhernovaiaiD· 2 shared Richard M. CamicioliiD· 2 shared Simon DuchesneiD· 2 shared D. Louis CollinsiD· 1 shared Douglas Lorne ArnoldiD· 1 shared For the CCNA Group· 1 shared

Activity by Year

2021-2022 · peak 2/yr

Co-Author Collaboration Network

9 top collaborators · 24 mutual co-author links

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3 shared papersNeuroscience

Josefina MaranzanoORCID

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3 papers

Recent·Most cited

Using Transfer Learning for Automated Microbleed Segmentation

Mahsa Dadar, Maryna Zhernovaia, Sawsan Mahmoud, Richard M. Camicioli, Josefina Maranzano, Simon Duchesne

This paper develops an automated microbleed segmentation tool using transfer learning on MRI data, achieving high sensitivity and specificity.

Frontiers in Neuroimaging · Aug 26, 2022Peer-reviewedNeuroscience

White Matter Hyperintensity Distribution Differences in Aging and Neurodegenerative Disease Cohorts

Mahsa Dadar, Sawsan Mahmoud, Maryna Zhernovaia, Richard M. Camicioli, Josefina Maranzano, Simon Duchesne, For the CCNA Group

The paper compares white matter hyperintensity (WMH) volumes across neurodegenerative disease groups and sexes, finding greater WMH burden in cognitively impaired groups and vascular disease subgroups.

NeuroImage Clinical · Jan 1, 2022Peer-reviewedNeuroscience

Diffusely abnormal white matter converts to T2 lesion volume in the absence of acute inflammation

Mahsa Dadar, Sawsan Mahmoud, Sridar Narayanan, D. Louis Collins, Douglas Lorne Arnold, Josefina Maranzano

This paper tracks the conversion of diffusely abnormal white matter to T2 lesions in MS patients, showing it occurs independently of inflammation and gadolinium enhancement.

Brain · Dec 16, 2021Peer-reviewedNeuroscience