Catalpol inhibits HHcy-induced EndMT in endothelial cells by modulating ROS/NF-κB signaling
Chengyan Wu, Rusheng Zhao, Shuangshuang Liu, Peixia Li, Huibing Liu, Libo Wang, Xuehui Wang
This study shows catalpol reduces HHcy-induced endothelial dysfunction by inhibiting EndMT through the ROS/NF-κB pathway in both in vitro and in vivo models.
BMC Cardiovascular Disorders · Aug 15, 2024Peer-reviewedMolecular Biology