Acute knockdown of extracellular matrix protein Tinagl1 disrupts heart laterality and pronephric cilia in zebrafish embryonic development
Hannah Neiswender, Ellen K. LeMosy
This study shows that knocking down Tinagl1 in zebrafish embryos causes heart laterality defects, ciliary abnormalities, and spinal cord issues, linking it to ciliopathy and signaling pathways.
bioRxiv · Jun 5, 2020 · PreprintDevelopmental Biology