Author

Tara E. McDonnell

University of TorontoORCID 0000-0001-6281-6732Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Assay:ChIP-seq· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Assay:whole-genome sequencing· 1 Organism:zebrafish· 1

Frequent Co-Authors

Francesca MedaiD· 1 shared Steven J. Deimling· 1 shared Vincent TropepeiD· 1 shared

Activity by Year

2026-2026 · peak 1/yr

Co-Author Collaboration Network

3 top collaborators · 3 mutual co-author links

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1 shared paperDevelopmental Biology

Francesca MedaORCID

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1 paper

Recent·Most cited

Loss of Ehmt2/G9a function in zebrafish is associated with global deficiency in H3K9 dimethylation, misregulated cell cycle dynamics, and embryonic developmental delay

Tara E. McDonnell, Francesca Meda, Steven J. Deimling, Vincent Tropepe

This study shows that loss of Ehmt2 function in zebrafish leads to reduced H3K9 dimethylation, delayed embryonic development, and altered cell cycle dynamics, but allows survival and fertility.

bioRxiv · Apr 7, 2026 · PreprintDevelopmental Biology