Author

Matteus Krappitz

Humboldt-Universität zu BerlinORCID 0000-0001-7764-4566Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Genetics· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 1 Approach:experimental assay· 1 Organism:human· 1 Approach:machine learning (classical)· 1 Organism:mouse· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

Jan HalbritteriD· 2 shared Kai‐Uwe EckardtiD· 2 shared Antonia IbeliD· 1 shared Axel Giesler· 1 shared Carlos Cordero· 1 shared Christian HinzeiD· 1 shared Christine KocksiD· 1 shared Dimitra PapaioannouiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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2 shared papersMolecular Biology

Jan HalbritterORCID

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2 papers

Recent·Most cited

Molecular BiologybioRxivPreprintMay 12, 2026

Cyst-type epithelial heterogeneity shapes therapeutic responsiveness in ADPKD

Johannes Roeles, Axel Giesler, Jessica Schmitz…·Christian Hinze

The paper identifies three epithelial cyst types in ADPKD using single-nucleus RNA sequencing and machine learning, showing their association with therapeutic response.

DOI 10.64898/2026.05.08.723838

humanscRNA-seq

𝕏 PostBluesky

GeneticsMolecular TherapyPeer-reviewedAug 21, 2025

In vivo base editing reduces liver cysts in autosomal dominant polycystic kidney disease

Antonia Ibel, Rishi Bhardwaj, Duygu Elif Yılmaz…·Michael M. Kaminski

This study shows that CRISPR base editing can correct pathogenic PKD1 variants in mice, reducing liver cysts and increasing polycystin-1 expression.

9 citationstop 3%

mouseexperimental assay

𝕏 PostBluesky