Author

Solbritt Rantapää‐Dahlqvist

Umeå UniversityORCID 0000-0001-8259-3863Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Genetic & Genomic Medicine· 1 Genetics· 1

Signature Assays, Organisms & Methods

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Research Focus:fine-mapping· 2 Assay:GWAS summary statistics· 2 Organism:human· 2 Approach:statistical model· 2 Research Focus:GWAS / association mapping· 1

Frequent Co-Authors

Javier J. MartiniD· 2 shared Lars KlareskogiD· 2 shared Peter K. GregerseniD· 2 shared Soumya RaychaudhuriiD· 2 shared Stephen EyreiD· 2 shared Tom W. J. HuizingaiD· 2 shared Yang LuoiD· 2 shared Aaron R. QuinlaniD· 1 shared

Activity by Year

2018-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 43 mutual co-author links

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2 shared papersGenetic & Genomic Medicine

Javier J. MartinORCID

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2 papers

Recent·Most cited

Genetic & Genomic MedicinemedRxivPreprintDec 1, 2021

Trans-ancestry genome-wide association study identifies novel genetic mechanisms in rheumatoid arthritis

Kazuyoshi Ishigaki, Saori Sakaue, Chikashi C. Terao…·Soumya Raychaudhuri

This study conducts a trans-ancestry GWAS of rheumatoid arthritis, identifying 124 loci, 34 novel, and shows improved polygenic risk score performance across populations.

13 citations

humanGWAS summary statistics

𝕏 PostBluesky

GeneticsNature GeneticsPeer-reviewedSep 11, 2018

Fine-mapping identifies causal variants for RA and T1D in DNASE1L3, SIRPG, MEG3, TNFAIP3 and CD28/CTLA4 loci

Harm-Jan Westra, Marta Martínez‐Bonet, Suna Önengüt-Gümüşcü…·Soumya Raychaudhuri

This study fine-maps RA and T1D loci outside the MHC, identifying causal variants and confirming their functional impact through allele-specific binding and enhancer activity.

148 citationstop 1%

humanGWAS summary statistics

𝕏 PostBluesky