Glynis L. Kolling

This study shows how environmental factors like nutrients, pH, and oxygen affect the evolution of antibiotic resistance in Pseudomonas aeruginosa.

This study shows that pathogenic Leptospira uses glucose under host-like temperatures, contradicting previous assumptions about its metabolic capabilities.

This study identifies metabolites from the soil bacterium Massilia that promote Leptospira growth, revealing a metabolic interaction between environmental bacteria and a pathogen.

The paper evaluates probiotic candidates for vaginal health using metabolic models and experimental assays, finding that D-lactic acid production, not metabolic similarity, drives inhibition of G. vaginalis.

The paper characterizes the functional metabolic diversity of Pseudomonas aeruginosa clinical isolates by analyzing genotype-phenotype correlations and genome-scale metabolic networks.

The paper develops a systems biology pipeline to design bacterial consortia that protect against severe Clostridioides difficile infection by targeting amino acid cross-feeding.

The paper identifies niche-specific metabolic targets in pathobionts by analyzing genome-scale metabolic networks and demonstrates their potential for targeted antimicrobial therapies.

This study introduces PROTEAN, a method to analyze metabolic capabilities of Lactobacillus strains, revealing site-specific differences in predicted metabolic production.