Author

A Keliris

University of AntwerpORCID 0000-0002-0576-3424Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Assay:imaging· 2 Organism:mouse· 2 Research Focus:cell-cell communication· 1 Approach:experimental assay· 1 Approach:statistical model· 1

Frequent Co-Authors

Annemie Van der LindeniD· 2 shared Georgios A. KelirisiD· 2 shared Inès R. H. Ben-Nejma· 2 shared Peter PonsaertsiD· 2 shared Jasmijn Daans· 1 shared Marleen VerhoyeiD· 1 shared Verdi VanreuseliD· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

7 top collaborators · 19 mutual co-author links

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2 shared papersNeuroscience

Annemie Van der LindenORCID

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2 papers

Recent·Most cited

Dynamics of brain-fluid circulation are altered in the mature-onset Tet-off APP mouse model of amyloidosis

Inès R. H. Ben-Nejma, A Keliris, Verdi Vanreusel, Peter Ponsaerts, Annemie Van der Linden, Georgios A. Keliris

This study shows altered brain fluid circulation dynamics in a mature-onset AD mouse model, linked to amyloid plaque burden and neuroinflammation.

bioRxiv · Mar 11, 2022 · PreprintNeuroscience

Increased soluble amyloid-beta causes early aberrant brain network hypersynchronisation in a mature-onset mouse model of amyloidosis

Inès R. H. Ben-Nejma, A Keliris, Jasmijn Daans, Peter Ponsaerts, Marleen Verhoye, Annemie Van der Linden, Georgios A. Keliris

This study shows that increased soluble amyloid-beta causes early brain network hypersynchronisation in a mature-onset mouse model of amyloidosis, with later hypoconnectivity and plaque formation.

Acta Neuropathologica Communications · Nov 14, 2019Peer-reviewedNeuroscience