Author

Sheila M. Bell

Cincinnati Children's Hospital Medical CenterORCID 0000-0002-1946-0969Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Developmental Biology· 1 Genetic & Genomic Medicine· 1

Signature Assays, Organisms & Methods

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Research Focus:gene regulatory network inference· 2 Approach:statistical model· 2 Assay:ATAC-seq· 1 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

Cheng-Lun NaiD· 2 shared Jeffrey A. WhitsettiD· 2 shared William J. ZachariasiD· 2 shared Alexander ZimprichiD· 1 shared Ángeles Montero· 1 shared Anna L. PeljtoiD· 1 shared Anthea CheungiD· 1 shared Anusha SridharaniD· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersGenetic & Genomic Medicine

Cheng-Lun NaORCID

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2 papers

Recent·Most cited

Genetic & Genomic MedicinemedRxivPreprintMay 19, 2026

Rare bi-allelic loss-of-function variants in the LRRK2 kinase cause interstitial lung disease

Tuğba Kalaycı, Kathryn A. Wikenheiser‐Brokamp, Sara Gomes…·Esther M. Sammler

This study identifies biallelic LRRK2 loss-of-function variants as a cause of interstitial lung disease, showing reduced LRRK2 protein and impaired lung function in affected individuals.

DOI 10.64898/2026.05.15.26352763

humanbulk RNA-seq

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Developmental BiologyNature CommunicationsPeer-reviewedSep 16, 2024

PRDM3/16 Regulate Chromatin Accessibility Required for NKX2-1 Mediated Alveolar Epithelial Differentiation and Function

Hua He, Sheila M. Bell, Ashley Kuenzi Davis…·Jeffrey A. Whitsett

This study shows PRDM3 and PRDM16 regulate chromatin accessibility to support NKX2-1-driven alveolar epithelial differentiation and function, with loss leading to respiratory failure and lineage infidelity.

21 citationstop 3%

mouseATAC-seq

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