Author

Praveen Bathini

Brigham and Women's HospitalORCID 0000-0002-4031-8975Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Approach:statistical model· 1

Frequent Co-Authors

Amrei FischeriD· 1 shared Cynthia Ann LemereiD· 1 shared David M. HoltzmaniD· 1 shared Edona Ballabani· 1 shared Elena ZenaroiD· 1 shared Eleonora TerrabuioiD· 1 shared Isabel Dupanloup· 1 shared Jens‐Ulrich RahfeldiD· 1 shared

Activity by Year

2020-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperNeuroscience

Amrei FischerORCID

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2 papers

Recent·Most cited

Early Binding of Anti-Amyloid Antibodies to CAA Drives Complement Activation, Inflammation and ARIA in Mice

Praveen Bathini, Stephan Schilling, Jens‐Ulrich Rahfeld, David M. Holtzman, Takaomi C. Saido, Cynthia Ann Lemere

This study investigates how early binding of anti-amyloid antibodies to cerebral amyloid angiopathy (CAA) drives complement activation, inflammation, and ARIA in Alzheimer's disease mouse models.

Molecular Neurodegeneration · Jun 26, 2026Peer-reviewedNeuroscience

Systemic inflammation causes microglial dysfunction with a mixed AD-like pathology.

Praveen Bathini, Isabel Dupanloup, Elena Zenaro, Eleonora Terrabuio, Amrei Fischer, Edona Ballabani, Marie‐Agnès Doucey, Lavinia Auber Alberi

This study shows that chronic systemic inflammation in mice leads to brain changes resembling mixed vascular and Alzheimer's disease pathology, including tauopathy and microglial dysfunction.

bioRxiv · Jul 27, 2020 · PreprintNeuroscience