Quercetin alters redox homeostasis, induces MAPK activation and promotes DNA damage in vGPCR-transformed endothelial cells
Gabriel Principe, Athina Maniscalchi, Silvina Tiburzi, Virginia Lezcano, Gabriela Alejandra Salvador, Verónica González Pardo
This study investigates how quercetin induces oxidative stress, MAPK activation, and DNA damage in vGPCR-transformed endothelial cells, suggesting its potential as a therapeutic agent for virus-associated cancers.
bioRxiv · Oct 6, 2026 · PreprintCancer Biology