Author

Giovanni Ristori

Sapienza University of RomeORCID 0000-0002-6598-6777Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:human· 4 Approach:statistical model· 4 Assay:GWAS summary statistics· 3 Research Focus:differential expression· 2 Research Focus:GWAS / association mapping· 2 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Renato UmetoniD· 4 shared Rosella MechelliiD· 4 shared Gianmarco BellucciiD· 3 shared Marco SalvettiiD· 3 shared Michela FerraldeschiiD· 3 shared Silvia RomanoiD· 3 shared Arianna FornasieroiD· 2 shared Carmela RomanoiD· 2 shared

Activity by Year

2019-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 42 mutual co-author links

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4 shared papersNeuroscience

Renato UmetonORCID

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4 papers

Recent·Most cited

GWAS-associated variants, non-genetic factors, and transient transcriptome in Multiple Sclerosis etiopathogenesis: a colocalization analysis

Renato Umeton, Gianmarco Bellucci, Rachele Bigi, Silvia Romano, Maria Chiara Buscarinu, Roberta Reniè, Virginia Rinaldi, Raffaella Pizzolato Umeton, Emanuele Morena, Carmela Romano, Rosella Mechelli, Marco Salvetti, Giovanni Ristori

This paper performs a colocalization analysis to explore how GWAS variants, non-genetic factors, and the transient transcriptome contribute to MS etiopathogenesis.

Scientific Reports · May 9, 2022Peer-reviewedNeuroscience

Circulating U13 small nucleolar RNA as a candidate biomarker for Huntington's disease

Silvia Romano, Carmela Romano, Martina Peconi, Alessia Fiore, Gianmarco Bellucci, Emanuele Morena, Fernanda Troili, Virginia Cipollini, Viviana Annibali, Simona Giglio, Rosella Mechelli, Michela Ferraldeschi, Liana Veneziano, Elide Mantuano, Gabriele Sani, , Giovanni Ristori

The paper identifies SNORD13 as a circulating biomarker for Huntington's disease, showing higher levels in HD patients compared to controls and correlating with disease status.

bioRxiv · Apr 22, 2022 · PreprintNeuroscience

Enrichment analysis of GWAS data in autoimmunity delineates the multiple sclerosis-Epstein Barr virus association

Rosella Mechelli, Renato Umeton, Virginia Rinaldi, Gianmarco Bellucci, Rachele Bigi, Daniela Francesca Angelini, Gisella Guerrera, Sundararajan Srinivasan, Silvia Romano, Maria Chiara Buscarinu, Raffaella Pizzolato Umeton, Eleni Anastasiadou, Pankaj Trivedi, Arianna Fornasiero, Michela Ferraldeschi, , Giovanni Ristori

This study identifies a link between Epstein Barr virus and multiple sclerosis through enrichment analysis of GWAS data and biological validation.

bioRxiv · Jun 6, 2021 · PreprintNeuroscience

Significant enrichment of Herpesvirus interactors in GWAS data suggests causal inferences for the association between Epstein Barr virus and multiple sclerosis

Rosella Mechelli, Renato Umeton, Sundararajan Srinivasan, Arianna Fornasiero, Michela Ferraldeschi, IMSGC and WTCCC2, Diego Centonze, Cinthia Farina, Marco Salvetti, Giovanni Ristori

The paper identifies significant enrichment of Herpesvirus interactors in GWAS data, suggesting a causal role for Epstein-Barr virus in multiple sclerosis.

bioRxiv · May 1, 2019 · PreprintNeuroscience