Author

Denise M. Inman

Northeast Ohio Medical UniversityORCID 0000-0002-8522-4112Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2

Frequent Co-Authors

Assraa Hassan Jassim· 1 shared August E. WoerneriD· 1 shared Aysha Ehsan· 1 shared Bindu KodatiiD· 1 shared Calvin D. BrooksiD· 1 shared Dorota L. StankowskaiD· 1 shared Drayanna FoxRun· 1 shared Hayden FlumeiD· 1 shared

Activity by Year

2020-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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1 shared paperNeuroscience

Assraa Hassan Jassim

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2 papers

Recent·Most cited

Involvement of Mitophagy in Endothelin-1 Mediated Neurodegeneration in Rodent Models of Glaucoma

Calvin D. Brooks, Bindu Kodati, Sanjna Prasad, Jacob Cunningham, Poojan Patel, Michael Mangan, Stacy M. Curry, Drayanna FoxRun, Aysha Ehsan, Ojasvi Arya, Hayden Flume, Kishor Kunwar, August E. Woerner, Denise M. Inman, Dorota L. Stankowska, Raghu R. Krishnamoorthy

This study investigates how endothelin-1 affects mitophagy in retinal ganglion cells and optic nerve in glaucoma models, finding differential responses in these regions.

bioRxiv · Jul 8, 2026 · PreprintNeuroscience

MCT2 Overexpression Rescues Metabolic Vulnerability and Protects Retinal Ganglion Cells in Two Models of Glaucoma

Mohammad Harun‐Or‐Rashid, Nathaniel Pappenhagen, Ryan D. Zubricky, Lucy Coughlin, Assraa Hassan Jassim, Denise M. Inman

This study shows that overexpressing MCT2 in glaucoma models preserves retinal ganglion cells and improves energy homeostasis by enhancing ATP production and mitochondrial function.

Neurobiology of Disease · May 15, 2020Peer-reviewedNeuroscience