Author

Hisashi Oishi

Nagoya City UniversityORCID 0000-0002-9988-6993Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Hisashi Oishi1 paper · Tohoku University

Biology Disciplines

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Genetic & Genomic Medicine· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Assay:protein structure· 1

Frequent Co-Authors

Mitsuharu HattoriiD· 2 shared Ajaz Ahmad BhatiD· 1 shared Akihiko SaitoiD· 1 shared Aljazi Al-Maraghi· 1 shared Ammira Al‐Shabeeb AkiliD· 1 shared Aya YoshidaiD· 1 shared Chris M. Danson· 1 shared Eisuke Okugawa· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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2 shared papersGenetic & Genomic Medicine

Mitsuharu HattoriORCID

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2 papers

Recent·Most cited

Genetic & Genomic MedicinemedRxivPreprintAug 17, 2024

The congenital multiple organ malformation syndrome, Ritscher-Schinzel syndrome is an endosomal recyclinopathy

Kohji Kato, Yosuke Nishio, Kirsty J. McMillan…·Peter J. Cullen

This study identifies RSS as a recyclinopathy caused by dysfunction in the Commander endosomal recycling pathway, linking it to multiple tissue malformations.

2 citations

NeuroscienceScientific ReportsPeer-reviewedMar 11, 2020

Physiological significance of proteolytic processing of Reelin revealed by cleavage-resistant Reelin knock-in mice

Eisuke Okugawa, Himari Ogino, Tomofumi Shigenobu…·Mitsuharu Hattori

This study shows that inhibiting Reelin cleavage enhances its signaling, leading to hippocampal layer disturbances and increased dendritic branching, suggesting Reelin cleavage is critical for normal brain development.

29 citations

mouseprotein structure