Author

Chuhan Chung

Gilead Sciences (United States)ORCID 0000-0003-2803-6745Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Genetic & Genomic Medicine· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Research Focus:GWAS / association mapping· 1 Assay:GWAS summary statistics· 1 Organism:human· 1

Frequent Co-Authors

Yevgeniy GindiniD· 2 shared Adam E. LockeiD· 1 shared Alyssa A. KriegermeieriD· 1 shared Amit V. KheraiD· 1 shared Andrew D. WellsiD· 1 shared Ann K. DalyiD· 1 shared Anurag VermaiD· 1 shared Aris BarasiD· 1 shared

Activity by Year

2022-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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2 shared papersMolecular Biology

Yevgeniy GindinORCID

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2 papers

Recent·Most cited

Molecular BiologyJournal of Lipid ResearchPeer-reviewedSep 24, 2022

Hepatic Deletion of X-box Binding Protein 1 in Farnesoid X Receptor Null Mice Leads to Enhanced Liver Injury

Xiaoying Liu, Mahmoud G. Khalafalla, Chuhan Chung…·Richard Green

This study investigates the role of the UPR in FXR null mice, finding that hepatic XBP1 activation provides protection against liver injury and tumor development.

8 citations

mousebulk RNA-seq

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Genetic & Genomic MedicineNature GeneticsPeer-reviewedJun 1, 2022

A trans-ancestry genome-wide association study of unexplained chronic ALT elevation as a proxy for nonalcoholic fatty liver disease with histological and radiological validation.

Marijana Vujković, Shweta Ramdas, Kimberly Lorenz…·Kyong‐Mi Chang

This study conducts a trans-ancestry GWAS using chronic ALT elevation as a proxy for NAFLD, identifies 77 significant loci, and validates 17 SNPs with histology and imaging data, revealing genetic links to metabolic and inflammatory traits.

205 citationstop 1%

humanGWAS summary statistics

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