Genetic & Genomic MedicinemedRxivPreprint
Short telomeres, but not accelerated epigenetic or proteomic age, increase fibrotic lung disease risk: Mendelian randomisation with pleiotropy dissection and independent replication
This study uses Mendelian randomisation to show that longer telomeres reduce fibrotic lung disease risk, independent of telomerase genes, while other ageing measures do not.
DOI 10.64898/2026.10.14.26364960