Author

Kira Breunig

The University of Texas at San Antonio Health Science CenterORCID 0009-0005-9004-2662Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Biophysics· 1 Genomics· 1

Signature Assays, Organisms & Methods

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Approach:biophysical modeling· 1 Organism:human· 1 Research Focus:protein folding & dynamics· 1 Assay:proteomics· 1

Frequent Co-Authors

David S. LibichiD· 2 shared Luiz O. F. PenalvaiD· 2 shared Adam KostiiD· 1 shared Antoine BaudiniD· 1 shared Corina Vinarov· 1 shared Gabriela D. A. GuardiaiD· 1 shared Hoang H. DinhiD· 1 shared Jennifer Chiou· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersBiophysics

David S. LibichORCID

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2 papers

Recent·Most cited

BiophysicsbioRxivPreprintMar 5, 2026

Intrinsically disordered SERBP1 regulates translation through topology-driven G-quadruplex recognition.

Antoine Baudin, Hoang H. Dinh, Kira Breunig…·David S. Libich

This paper identifies SERBP1 as a G4-binding protein that regulates translation through topology-driven recognition of G-quadruplex structures in RNA, with implications for cancer therapy.

DOI 10.64898/2026.03.03.709370

humanproteomics

𝕏 PostBluesky

GenomicseLifePeer-reviewedFeb 12, 2025

SERBP1 interacts with PARP1 and is present in PARylation-dependent protein complexes regulating splicing, cell division, and ribosome biogenesis

Kira Breunig, Xiufen Lei, Mauro Montalbano…·Luiz O. F. Penalva

The paper identifies SERBP1 as a protein involved in PARylation-dependent complexes that regulate splicing, cell division, and ribosome biogenesis, with implications for neurodegenerative diseases.

5 citations

𝕏 PostBluesky