Author

A. Bantan

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:clustering· 1 Approach:deep learning· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:human· 1

Frequent Co-Authors

B. Paiva· 2 shared B. Saez· 2 shared D. Gomez-Cabrero· 2 shared E. Muinos-Lopez· 2 shared F. Prosper· 2 shared I. Cenzano· 2 shared I. Sancho Gonzalez· 2 shared J. A. Martinez-Climent· 2 shared

Activity by Year

2024-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersCancer Biology

B. Paiva

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Bone Marrow Spatial Transcriptomics Reveals a Myeloma Cell Architecture with Dysfunctional T-Cell Distribution, Neutrophil Traps, and Inflammatory Signaling

L. Sudupe, E. Muinos-Lopez, A. R. Lopez-Perez, A. Vilas-Zornoza, S. Sarvide, P. Ripalda-Cemborain, P. Aguirre-Ruiz, P. San Martin-Uriz, M. Larrayoz, L. Alvarez-Gigli, M. Abengozar-Muela, I. Cenzano, M. Cocera, J. Ruiz, I. Sancho Gonzalez, , F. Prosper

This study uses spatial transcriptomics to analyze bone marrow in MM mouse models and patients, revealing altered T-cell distribution, neutrophil traps, and inflammatory signaling in MM.

bioRxiv · Jul 3, 2024 · PreprintCancer Biology

Stromal and Endothelial Transcriptional Changes during progression from MGUS to Myeloma and after Treatment Response

I. Cenzano, M. Cocera, M. Larrayoz, L. Campos-Dopazo, S. Sanz, A. Bantan, A. Vilas, P. San-Martin, P. Aguirre-Ruiz, D. Alignani, A. Lopez, I. Sancho Gonzalez, J. Ruiz, P. Ripalda-Cemborain, M. Abengozar-Muela, , F. Prosper

This study identifies transcriptional changes in stromal and endothelial cells during MGUS to myeloma progression and treatment response, revealing a tumor-permissive BME signature that is modulated by therapy.

bioRxiv · Apr 24, 2024Cancer Biology