Author

A. C. Telang

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Pathology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Approach:genetic perturbation / CRISPR· 1 Organism:mouse· 1

Frequent Co-Authors

A. Al-Suraimi· 1 shared A. Soofi· 1 shared E. A. Otto· 1 shared G. R. Dressler· 1 shared J. A. Beamish· 1 shared J. M. Weinberg· 1 shared J. T. Ference-Salo· 1 shared M. C. McElliott· 1 shared

Activity by Year

2023-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperPathology

A. Al-Suraimi

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1 paper

Recent·Most cited

Pax Protein Depletion in Proximal Tubules Triggers Conserved Mechanisms of Resistance to Acute Ischemic Kidney Injury and Prevents Transition to Chronic Kidney Disease

J. A. Beamish, A. C. Telang, M. C. McElliott, A. Al-Suraimi, M. Chowdhury, J. T. Ference-Salo, E. A. Otto, R. Menon, A. Soofi, J. M. Weinberg, S. R. Patel, G. R. Dressler

This study shows that deleting Pax2 and Pax8 in proximal tubules reduces acute kidney injury severity and prevents chronic disease by inducing protective gene expression similar to preconditioning.

bioRxiv · Oct 3, 2023 · PreprintPathology