H3.1K27me1 maintains transcriptional silencing and genome stability by preventing GCN5-mediated histone acetylation
J. Dong, C. LeBlanc, A. Poulet, B. Mermaz, G. Villarino, K. M. Webb, V. Joly, J. Mendez, P. Voigt, Y. Jacob
This study identifies GCN5 as a mediator of transcriptional de-repression and genomic instability in plants lacking H3.1K27me1, revealing its role in maintaining genome stability through histone acetylation regulation.
bioRxiv · Jul 17, 2020Plant Biology