Author

A. W. Horne

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Immunology· 2

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

C. Hogg· 2 shared E. Greaves· 2 shared M. Rosser· 2 shared P. Dhami· 2 shared A. O. Cuff· 1 shared A. Senior· 1 shared D. Soong· 1 shared E. T. Courtois· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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2 shared papersImmunology

C. Hogg

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2 papers

Recent·Most cited

Single-cell analysis identifies distinct macrophage phenotypes associated with pro-disease and pro-resolving functions in the endometriotic niche

Y. Henlon, K. Panir, I. McIntyre, C. Hogg, P. Dhami, A. O. Cuff, A. Senior, N. Moolchandani-Adwani, E. T. Courtois, A. W. Horne, M. Rosser, S. Ott, E. Greaves

The paper identifies distinct macrophage phenotypes in endometriosis using single-cell analysis, linking them to disease-promoting and resolving functions.

bioRxiv · Mar 7, 2024 · PreprintImmunology

Macrophages can inhibit and enhance endometriosis depending on their origin

C. Hogg, P. Dhami, M. Rosser, M. Mack, D. Soong, J. W. Pollard, S. J. Jenkins, A. W. Horne, E. Greaves

This study shows that macrophages in endometriosis lesions can either promote or suppress disease depending on their origin, with eutopic endometrial macrophages being pro-endometriosis and recruited monocyte-derived macrophages being anti-endometriosis.

bioRxiv · Apr 30, 2020Immunology