Author

A. Wenzel

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Organism:human· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Approach:deep learning· 1 Research Focus:differential expression· 1 Assay:proteomics· 1

Frequent Co-Authors

B. T. Porse· 2 shared M. B. Schuster· 2 shared A. S. Wilhelmson· 1 shared B. Furtwaengler· 1 shared C. Gentil· 1 shared D. Barmpouri· 1 shared E. Heyes· 1 shared E. M. Schoof· 1 shared

Activity by Year

2023-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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2 shared papersCell Biology

B. T. Porse

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2 papers

Recent·Most cited

Mapping the human hematopoietic stem and progenitor cell hierarchy through integrated single-cell proteomics and transcriptomics

B. Furtwaengler, N. Uresin, S. Richter, M. B. Schuster, D. Barmpouri, H. Holze, A. Wenzel, K. Gronbaek, K. Theilgaard-Moench, F. J. Theis, E. M. Schoof, B. T. Porse

The paper integrates single-cell proteomics and transcriptomics to study human hematopoietic stem and progenitor cells, identifying protein markers and dynamics not captured by mRNA alone.

bioRxiv · Jul 5, 2024 · PreprintCell Biology

TET2 lesions enhance the aggressiveness of CEBPA-mutant AML by rebalancing GATA2 expression

E. Heyes, A. S. Wilhelmson, A. Wenzel, G. Manhart, T. Eder, M. B. Schuster, E. Rzepa, S. Pundhir, T. D'Altri, C. Gentil, M. Meggendorfer, J. Schwaller, T. Haferlach, F. Grebien, B. T. Porse

This study identifies GATA2 as a target of the CEBPA-TET2 mutational axis in AML, showing how TET2 lesions rebalance GATA2 expression and enhance disease aggressiveness.

bioRxiv · Mar 28, 2023Cancer Biology