Author

B. C. Wilson

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Pathology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Research Focus:cell type annotation· 2 Assay:imaging· 2 Research Focus:chromatin & epigenetic regulation· 1 Approach:deep learning· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Approach:microscopy & imaging analysis· 1

Frequent Co-Authors

A. Golaraei· 2 shared E. Zurauskas· 2 shared K. Mirsanaye· 2 shared L. Kontenis· 2 shared V. Barzda· 2 shared Y. Kamaliddin· 2 shared A. Annibali· 1 shared A. Hodgkinson· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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2 shared papersPathology

A. Golaraei

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Author profile →

3 papers

Recent·Most cited

Unsupervised determination of lung tumor margin with widefield polarimetric second-harmonic generation microscopy

K. Mirsanaye, L. U. Castano, Y. Kamaliddin, A. Golaraei, L. Kontenis, E. Zurauskas, R. Navab, K. Yasufuku, M.-S. Tsao, B. C. Wilson, V. Barzda

This paper uses P-SHG microscopy and unsupervised clustering to identify tumor margins in lung tissue by analyzing ECM collagen structure.

bioRxiv · Jul 25, 2022Pathology

Machine learning-enabled cancer diagnostics with widefield polarimetric second-harmonic generation microscopy

K. Mirsanaye, L. Uribe Castano, Y. Kamaliddin, A. Golaraei, R. Augulis, L. Kontenis, S. J. Done, E. Zurauskas, B. C. Wilson, V. Barzda

This paper uses machine learning with polarimetric second-harmonic generation microscopy to classify breast tissue as cancerous or normal with high accuracy.

bioRxiv · May 26, 2021Pathology

Intellectual disability-associated factor Zbtb11 cooperates with NRF-2/GABP to control mitochondrial function

B. C. Wilson, L. Boehme, A. Annibali, A. Hodgkinson, T. S. Carroll, R. J. Oakey, V. C. Seitan

This study identifies Zbtb11 as a mitochondrial regulator that interacts with NRF-2/GABP to maintain mitochondrial function and shows its disruption leads to intellectual disability.

bioRxiv · Dec 13, 2019Cell Biology