Author

C. D. Van Raamsdonk

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Research Focus:gene regulatory network inference· 2 Organism:mouse· 2 Research Focus:cell-cell communication· 1 Approach:diffusion model· 1

Frequent Co-Authors

A. Haage· 1 shared A. Luty· 1 shared A. N. Longakit· 1 shared F. Jain· 1 shared G. Tanentzapf· 1 shared O. Urtatiz· 1 shared R. Farag· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

7 top collaborators · 9 mutual co-author links

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1 shared paperCancer Biology

A. Haage

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2 papers

Recent·Most cited

Cancer BiologybioRxivPreprintMay 24, 2023

BAP1 deficient human and mouse uveal melanomas up-regulate a shared EMT pathway

R. Farag, F. Jain, A. N. Longakit…·C. D. Van Raamsdonk

The paper investigates BAP1 loss in uveal melanomas, finding it reduces lung lesions but increases liver lesions and identifies a shared EMT pathway in human and mouse tumors.

DOI 10.1101/2023.05.24.542173

mousehuman

𝕏 PostBluesky

Cancer BiologybioRxivJul 26, 2021

Crosstalk with keratinocytes causes GNAQ oncogene specificity in melanoma

O. Urtatiz, A. Haage, G. Tanentzapf, C. D. Van Raamsdonk

The paper shows that epidermal crosstalk impairs GNAQQ209L-expressing melanocytes, explaining GNAQ oncogene specificity in cutaneous melanoma.

DOI 10.7554/elife.71825

mousebulk RNA-seq

𝕏 PostBluesky