Author

Chris Cotsapas

Primary ORCID profile (0000-0002-7772-5910 · 1 paper)Unclaimed ORCID profile · follow to get notified of new preprints

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Chris CotsapasiD1 paper

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:statistical model· 4 Organism:human· 3 Research Focus:eQTL / molecular QTL mapping· 2 Research Focus:gene regulatory network inference· 2 Assay:GWAS summary statistics· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Research Focus:GWAS / association mapping· 1

Frequent Co-Authors

JinMyung Choi· 2 shared Parisa Shooshtari· 2 shared Alexandra Casparino· 1 shared Barbara Stranger· 1 shared Benjamin Neale· 1 shared Benjamin Raby· 1 shared Benjamin Voight· 1 shared Boel Brynedal· 1 shared

Activity by Year

2015-2016 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 15 mutual co-author links

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2 shared papersGenetics

JinMyung Choi

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4 papers

Recent·Most cited

Large-scale trans-eQTLs affect hundreds of transcripts and mediate patterns of transcriptional co-regulation

Boel Brynedal, JinMyung Choi, Towfique Raj, Robert Bjornson, Barbara Stranger, Benjamin Neale, Benjamin Voight, Chris Cotsapas

The paper identifies trans-eQTLs that affect multiple transcripts and mediate transcriptional co-regulation across populations.

bioRxiv · May 31, 2016Genetics

Integrative genetic and epigenetic analysis uncovers regulatory mechanisms of autoimmune disease

Parisa Shooshtari, Hailiang Huang, Chris Cotsapas

This study identifies regulatory regions and genes associated with autoimmune diseases using integrative genetic and epigenetic analysis of public data.

bioRxiv · May 19, 2016Genetics

Shared effect modeling reveals that a fraction of autoimmune disease associations are consistent with eQTLs in three immune cell types

Sung Chun, Alexandra Casparino, Nikolaos Patsopoulos, Damien Croteau-Chonka, Benjamin Raby, Philip De Jager, Shamil Sunyaev, Chris Cotsapas

This study identifies that about 25% of autoimmune disease loci have eQTLs driven by the same genetic effect using a shared effect modeling approach.

bioRxiv · May 12, 2016Genetics

Network analysis of genome-wide selective constraint reveals a gene network active in early fetal brain intolerant of mutation

Jinmyung Choi, Parisa Shooshtari, Kaitlin E Samocha, Mark J Daly, Chris Cotsapas

The paper identifies a gene network under strong selective constraint in early fetal brain development and shows it is enriched for genes mutated in neurological disorders.

bioRxiv · Mar 29, 2015Genetics