Author

D. A. Gaykalova

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:cell-cell communication· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Assay:scRNA-seq· 2 Assay:ATAC-seq· 1 Assay:bulk RNA-seq· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

E. J. Fertig· 2 shared L. T. Kagohara· 2 shared S. Yegnasubramanian· 2 shared A. Gupta· 1 shared B. J. Christmas· 1 shared C. Rafie· 1 shared D. N. Sidiropoulos· 1 shared E. Bigelow· 1 shared

Activity by Year

2019-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersImmunology

E. J. Fertig

Mutual co-authors in cluster

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2 papers

Recent·Most cited

ImmunologybioRxivPreprintJan 29, 2021

Phenotypic shifts of tumor associated macrophages and STAT3 mediated suppression of myeloid derived suppressor cells drive sensitization of HER2+ tumor immunity.

D. N. Sidiropoulos, C. Rafie, B. J. Christmas…·E. T. Roussos-Torres

The paper uses single-cell RNA-sequencing to show entinostat shifts tumor microenvironment cell states, reducing immunosuppression and enhancing immune checkpoint inhibitor responses in HER2+ breast tumors.

DOI 10.1101/2021.01.29.428708

mousescRNA-seq

𝕏 PostBluesky

Cancer BiologybioRxivNov 6, 2019

Integrated single cell and bulk multi-omics reveals heterogeneity and early changes in pathways associated with cetuximab resistance in HNSCC sensitive cell lines

L. T. Kagohara, F. Zamuner, M. Considine…·E. J. Fertig

The paper uses single cell and bulk multi-omics to study early resistance mechanisms in HNSCC cells treated with cetuximab, identifying TFAP2A and EMT as independent pathways.

DOI 10.1038/s41416-020-0851-5

humanscRNA-seq

𝕏 PostBluesky