Author

D. Hoak

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Pathology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

J. H. Jonason· 1 shared J. P. Ketz· 1 shared M. Chang· 1 shared M. J. Hilton· 1 shared M. J. Zuscik· 1 shared S. E. Catheline· 1 shared

Activity by Year

2018-2018 · peak 1/yr

Co-Author Collaboration Network

6 top collaborators · 15 mutual co-author links

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1 shared paperPathology

J. H. Jonason

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1 paper

Recent·Most cited

Chondrocyte-specific RUNX2 Overexpression Causes Chondrodysplasia During Development, but is Not Sufficient to Induce OA-like Articular Cartilage Degeneration in Adult Mice Without Injury

S. E. Catheline, D. Hoak, M. Chang, J. P. Ketz, M. J. Hilton, M. J. Zuscik, J. H. Jonason

This study shows that chondrocyte-specific RUNX2 overexpression during development causes chondrodysplasia but does not induce OA-like cartilage degeneration in adult mice without injury, though it accelerates OA progression after trauma.

bioRxiv · Nov 14, 2018 · PreprintPathology