Author

I. Stillman

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Pathology· 1

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Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

B. K. Subramanian· 1 shared C. Perez· 1 shared H. Higgs· 1 shared J. Chun· 1 shared J. Schlondorff· 1 shared M. Pollak· 1 shared P. Yan· 1 shared S. Alper· 1 shared

Activity by Year

2019-2019 · peak 1/yr

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8 top collaborators · 28 mutual co-author links

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1 shared paperPathology

B. K. Subramanian

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1 paper

Recent·Most cited

Proteolytic program-dependent functions are impaired in INF2-mediated focal segmental glomerulosclerosis.

B. K. Subramanian, J. Chun, C. Perez, P. Yan, I. Stillman, H. Higgs, S. Alper, J. Schlondorff, M. Pollak

This study identifies a proteolytic cleavage mechanism in INF2 that affects its function in podocytes, leading to impaired localization and contributing to FSGS.

bioRxiv · Jan 26, 2019 · PreprintPathology