Author

Jacob Forstrom

Barrow Neurological Institute

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Genetics· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Organism:Drosophila· 2 Approach:experimental assay· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Michael C. KrueriD· 2 shared Sara A. LewisiD· 2 shared Sergio R. Padilla-LopeziD· 2 shared Allan BayatiD· 1 shared Ebba AlkhunaiziiD· 1 shared Frédéric BilaniD· 1 shared Gwenaël Le GuyaderiD· 1 shared Hilary J. VernoniD· 1 shared

Activity by Year

2023-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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2 shared papersNeuroscience

Michael C. KruerORCID

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2 papers

Recent·Most cited

eIF2α phosphorylation evokes dystonia-like movements with D2-receptor and cholinergic origin and abnormal neuronal connectivity

Sara A. Lewis, Jacob Forstrom, Jennifer Renee Tavani, Robert Schafer, Zach Tiede, Sergio R. Padilla-Lopez, Michael C. Kruer

This study shows that altering eIF2α phosphorylation in Drosophila models induces dystonia-like movements with cholinergic and D2-receptor origins and altered neuronal connectivity.

bioRxiv · May 14, 2024 · PreprintNeuroscience

AGAP1-associated endolysosomal trafficking abnormalities link gene-environment interactions in a neurodevelopmental disorder with incomplete penetrance

Sara A. Lewis, Somayeh Bakhtiari, Jacob Forstrom, Allan Bayat, Frédéric Bilan, Gwenaël Le Guyader, Ebba Alkhunaizi, Hilary J. Vernon, Sergio R. Padilla-Lopez, Michael C. Kruer

The paper describes three patients with AGAP1 deletion variants and uses a Drosophila model to identify endolysosomal trafficking defects and stress response abnormalities linked to neurodevelopmental disorders.

Disease Models & Mechanisms · Jul 20, 2023Peer-reviewedGenetics