Identification of endogenous Adenomatous polyposis coli interaction partners and β-catenin-independent targets by proteomics
O. Popow, M. Tatham, J. Paulo, A. Rojaa, N. Loyer, I. P. Newton, J. Januschke, K. Haigis, I. Nathke
The paper identifies APC interaction partners and β-catenin-independent targets using proteomics, revealing MINK1 as a potential substrate of an alternative APC destruction complex.
bioRxiv · Feb 1, 2018Cancer Biology