Author

L. Fol

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:chromatin & epigenetic regulation· 1 Assay:DNA methylation· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Approach:statistical model· 1

Frequent Co-Authors

A. Fasciani· 1 shared A. Turdo· 1 shared a. Zippo· 1 shared F. Demichelis· 1 shared G. D Amato· 1 shared M. Botteon· 1 shared M. Gaggianesi· 1 shared M. Todaro· 1 shared

Activity by Year

2024-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperCancer Biology

A. Fasciani

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1 paper

Recent·Most cited

ANP32E drives vulnerability to ATR inhibitors by inducing R-loops-dependent Transcription Replication Conflicts in Triple Negative Breast Cancer

S. Lago, V. Poli, L. Fol, M. Botteon, A. Fasciani, A. Turdo, M. Gaggianesi, M. Todaro, Y. Ciani, G. D Amato, F. Demichelis, a. Zippo

The paper identifies ANP32E as a driver of vulnerability to ATR inhibitors in triple-negative breast cancer by promoting R-loop-dependent transcription replication conflicts and genomic instability.

bioRxiv · Mar 25, 2024Cancer Biology