Author

M. Kapustina

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

A. Hertel-Wulff· 1 shared B. Wall· 1 shared D. I. Stuart· 1 shared D. Li· 1 shared E. E. Fry· 1 shared H. Feng· 1 shared H. M. E. Duyvesteyn· 1 shared J. A. Duncan· 1 shared

Activity by Year

2022-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperMicrobiology

A. Hertel-Wulff

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2 papers

Recent·Most cited

Cell surface excess is essential for protrusions and motility in 3D matrix

M. Kapustina, D. Li, J. Zhu, B. Wall, V. Weinreb, R. E. Cheney

This study shows that cell surface excess (CSE) is crucial for protrusions and motility in 3D collagen matrices, with microtubules regulating its storage and release.

bioRxiv · Aug 12, 2022 · PreprintCell Biology

Nonlytic cellular release of hepatitis A virus requires dual capsid recruitment of the ESCRT-associated Bro1 domain proteins HD-PTP and ALIX

T. Shirasaki, H. Feng, H. M. E. Duyvesteyn, W. G. Fusco, K. L. McKnight, L. Xie, M. Boyce, S. Kumar, R. Barouch-Bentov, O. Gonzalez-Lopez, R. McNamara, L. Wang, A. Hertel-Wulff, X. Chen, S. Einav, , S. M. Lemon

This study identifies a viral export signal in hepatitis A virus that requires dual recruitment of HD-PTP and ALIX for nonlytic release in extracellular vesicles.

bioRxiv · Apr 26, 2022Microbiology