Author

M. Rudelius

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Organism:human· 1 Assay:imaging· 1 Organism:mouse· 1

Frequent Co-Authors

A. Dueck· 1 shared B. Walzog· 1 shared C. D. Strobl· 1 shared C. Guo· 1 shared C. Ludwig· 1 shared C. Nerlov· 1 shared C. Scheiermann· 1 shared C. Schulz· 1 shared

Activity by Year

2022-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperImmunology

A. Dueck

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

ARID1A Mutations Protect Follicular Lymphoma from FAS-dependent Immune Surveillance by Reducing RUNX3/ETS1-Driven FAS-Expression

M. Antoniolli, M. Solovey, C. D. Strobl, D. Bararia, W. D. Keay, J. A. Hildebrand, L. Adolph, M. Heide, V. Passerini, T. Freyholdt, L. Wange, W. Enard, S. Thieme, H. Blum, M. Rudelius, , O. Weigert

This study identifies ARID1A mutations in follicular lymphoma that reduce FAS expression, enabling immune evasion by disrupting RUNX3/ETS1 regulation.

bioRxiv · Dec 12, 2023 · PreprintCancer Biology

Plasmacytoid dendritic cells regulate megakaryocyte and platelet homeostasis

F. Gaertner, H. Ishikawa-Ankerhold, S. Stutte, W. Fu, C. Guo, J. Weitz, A. Dueck, Z. Zhang, D. van den Heuvel, V. Fumagalli, M. Lorenz, L. von Baumgarten, K. Stark, T. Straub, S. von Stillfried, , S. Massberg

This study identifies plasmacytoid dendritic cells (pDCs) as regulators of megakaryopoiesis and platelet homeostasis, showing their role in maintaining steady-state and stress conditions, and their involvement in SARS-CoV2-induced thrombosis.

bioRxiv · May 31, 2022 · PreprintImmunology