Author

M.-A. Bringer

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:E. coli· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

G. Demarre· 2 shared N. Barnich· 2 shared O. Espeli· 2 shared S. Rimsky· 2 shared V. Prudent· 2 shared A. Morillon· 1 shared A. Ravet· 1 shared E. Rousseau· 1 shared

Activity by Year

2019-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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2 shared papersMicrobiology

G. Demarre

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2 papers

Recent·Most cited

The Crohn's disease-related AIEC strain LF82 assembles a biofilm-like matrix to protect intracellular microcolonies from phagolysosomal attack

V. Prudent, G. Demarre, E. Vazeille, M. Wery, A. Ravet, N. Quenech Du, J. Dauverd Girault, M.-A. Bringer, M. Descrimes, N. Barnich, S. Rimsky, A. Morillon, O. Espeli

This study shows that the AIEC strain LF82 forms a biofilm-like matrix to protect intracellular bacterial communities from phagolysosomal attack in macrophages.

bioRxiv · Mar 31, 2020 · PreprintMicrobiology

The Crohn s disease-associated Escherichia coli strain LF82 rely on SOS and stringent responses to survive, multiply and tolerate antibiotics within macrophages.

G. Demarre, V. Prudent, H. Schenk, E. Rousseau, M.-A. Bringer, N. Barnich, G. Tran Van Nhieu, S. Rimsky, S. De Monte, O. Espeli

This study investigates how AIEC LF82 survives and multiplies in macrophages, revealing phenotypic switches and antibiotic tolerance mechanisms.

bioRxiv · Feb 27, 2019Microbiology