Myeloid Cell States in Influenza-Associated Pulmonary Aspergillosis Are Shaped by Iron Overload and Metabolic Reprogramming
Madeleine S. Grau, Brian P. Jackson, Carol S. Ringelberg, Fred Kolling, George S. Deepe, Robert A. Cramer, Joshua J. Obar
This study identifies iron overload and metabolic reprogramming in myeloid cells as drivers of susceptibility to Aspergillus fumigatus in influenza-associated pulmonary aspergillosis.
bioRxiv · Apr 25, 2026 · PreprintImmunology