Author

N. Haq

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cell Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

C. Schmidt-Hieber· 1 shared E. Forsythe· 1 shared F. J. Sialana· 1 shared G. A. Bewick· 1 shared G. Lubec· 1 shared J. P. Heller· 1 shared J. Pearson· 1 shared K. Toczyska· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperCell Biology

C. Schmidt-Hieber

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Adherent reformed islets: a long-term primary cell-based platform for exploring mouse and human islet biology.

N. Haq, K. Toczyska, M. Wilson, M. Jacobs, Y. Lei, M. Zhao, J. Pearson, S. Persaud, T. Pullen, G. A. Bewick

This study develops a long-term primary cell-based platform called reformed islets to study mouse and human islet biology, showing they retain key physiological and functional features of native islets.

bioRxiv · Nov 22, 2023 · PreprintPhysiology

Loss of Bardet-Biedl syndrome proteins causes synaptic aberrations in principal neurons

N. Haq, C. Schmidt-Hieber, F. J. Sialana, L. Ciani, J. P. Heller, M. Stewart, L. Bentley, S. Wells, R. J. Rodenburg, P. M. Nolan, E. Forsythe, M. C. Wu, G. Lubec, M. Hausser, P. Beales, S. Christou-Savina

This study shows that loss of Bardet-Biedl syndrome proteins causes synaptic dysfunction in principal neurons, contributing to cognitive impairment in BBS patients.

bioRxiv · Mar 18, 2019Cell Biology