Author

Nicole Kreiter

German Center for Neurodegenerative Diseases (DZNE) Dresden

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:trajectory inference· 1

Frequent Co-Authors

Andreas HermanniD· 2 shared Arun Pal· 2 shared Jared L. SterneckertiD· 2 shared Alexander StorchiD· 1 shared Benedikt Kretner· 1 shared Florian WegneriD· 1 shared Hannes GlaßiD· 1 shared Julia Japtok· 1 shared

Activity by Year

2018-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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2 shared papersNeuroscience

Andreas HermannORCID

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2 papers

Recent·Most cited

High content live profiling reveals concomitant gain and loss of function pathomechanisms in C9ORF72 amyotrophic lateral sclerosis.

Arun Pal, Benedikt Kretner, Masin Abo-Rady, Hannes Glaß, Marcel Naumann, Julia Japtok, Nicole Kreiter, Tobias Maria Böckers, Jared L. Sterneckert, Andreas Hermann

This study shows that C9ORF72 ALS pathogenesis involves both gain and loss of function mechanisms causing axonal organelle trafficking impairment and DNA damage.

bioRxiv · Apr 15, 2020 · PreprintNeuroscience

Age-dependent neurodegeneration and organelle transport deficiencies in mutant TDP43 patient-derived neurons are independent of TDP43 aggregation

Nicole Kreiter, Arun Pal, Xenia Lojewski, Philippe Corcia, Maximilian Naujock, Peter Reinhardt, Jared L. Sterneckert, Susanne Petri, Florian Wegner, Alexander Storch, Andreas Hermann

This study shows that TDP43 mutations cause motor neuron degeneration in ALS through mechanisms unrelated to TDP43 aggregation or mislocalization.

Neurobiology of Disease · Apr 6, 2018Peer-reviewedNeuroscience